~ Open Letter to All Bacterial Infected People ~
* * *
2010-05-27
13:23:18
Dear Global Citizens,
It is well known around the world and an accepted science that Tripalmitoyl cysteine (Pam3Cys), the LYMErix and HIV vaccines and the failed lipoprotein vaccine trials for Tuberculosis cause immunosuppression via the mechanisms described in the Pam3Cys PPT presentation you can view here:
http://www.lymecryme.com/rich_text_21.html
Along with the revelation that Epstein-Barr virus also has TLR2 agonist ligands, http://www.ncbi.nlm.nih.gov/pubmed?term=ebv%5bAll%20Fields%5d%20AND%20tlr2%5bAll%20Fields%5d&cmd=DetailsSearch
Some folks with Chronic Lyme are saying,
"Where do we go from here?"
If it is true that the National Institute of Neurological Disorders and Stroke's, Roland Martin was treating Lyme-MS as Lyme and MS (with both ceftriaxone and MS meds), and that this is truly the evidence-based medicine:
http://www.ncbi.nlm.nih.gov/pubmed/16783164
A) IDSA's Treatment Failure reports
- The Biology of Parasitic Spirochetes:
Allen Steere on treatment failure after multiple courses of antibiotics:
Allen Steere on treatment failure:
Congenital Lyme post-treatment of the mother, Allen Steere:
Persistence of Borrelia burgdorferi Following Antibiotic Treatment in Mice"
B) Ben Luft's recent claim
- "These results extended previous studies with ceftriaxone, indicating that antibiotic treatment is unable to clear persisting spirochetes, which remain viable and infectious, but are nondividing or slowly dividing."
C) CDC says "Bb is intracellular"
- "Cytopathic effects were not observed following infection of these cell lines with B. burgdorferi, and internalized spirochetes were found to be viable. Invasion of neural cells by B. burgdorferi provides a putative mechanism for the organism to avoid the host's immune response while potentially causing functional damage to neural cells during infection of the CNS."
D) Mark Klempner reported that "Bb is intracellular and thereby resistant to ceftriaxone,"
E) Brian Fallon discovered that indeed:
- Lyme is Relapsing Fever and the longer term treatment past the 30 days of Mark Klempner's standard of care results in remission, but then relapse (making Lyme FUNCTIONALLY identical to Relapsing Fever in addition to it being Taxonomically the same disease):
F) and now that Gary Wormser and Mark Klempner have re-admitted that Bb antigens like OspA cause anti-brain/anti-nerve antibodies
http://www.ncbi.nlm.nih.gov/pubmed/20227484
Patients are wondering,
"Where do I/we stand?"
"How should I/we be treated?"
Because Lyme Borreliosis is the "New Great Imitator," Syphilis is the "Great Imitator" and OspA vaccination was the "Greatest Imitator," and because we know the major clues to the crimes were the publications of:
1) NCI's and the US Army's Paul Duray "Immature, badly cloned, Epstein-Barr-like immortalized B cells,"
2) Justin Radolf [TLR2 agonism (with OspA) described how OspA-like molecules are responsible for the downregulation of HLA molecules (or that after a time, no antibodies are produced):
3) and the Korean Chemists who revealed that LYMErix was the same as HIV vaccine antigens gp120/41 (Pam3Cys)
How do we deal with all the diseases that we have, since it clearly looks like we have a form of OspA-induced Acquired Immune Deficiency and incompetent B cells? We are not talking only about activated Epstein-Barr, cytomegalovirus and the other herpes viruses, and we're not only talking about incompetent B cells, we are also talking about, in many Lyme cases, the tick-borne co-infections like the Ehrlichias and Babesia for which still, today, there are no accurate blood tests.
We can not ask ILADS.org because they have not stayed current with the science and of course the recommendations of IDSociety.org are out of the question, given that because the IDSA:
a) lied about the outcomes of their OspA vaccines,
b) falsified the testing for Lyme (Dearborn) in order to falsify their vaccines outcomes, and
c) never reported to the NIH or especially NIAID - the division headed by Edward McSweegan's boss, Anthony Fauci. - that they could not even read their Western Blots in OspA vaccinated people:
LYMErix results (76% "safe and effective"):
http://content.nejm.org/cgi/content/abstract/339/4/209
ImmuLyme results (92% "safe and effective"):
http://content.nejm.org/cgi/content/abstract/339/4/216
But we could NOT actually read our Western Blots in this OspA vaccines trials.
http://jcm.asm.org/cgi/reprint/35/1/233?view=long&pmid=8968914
%2Fnetahtml%2FPTO%2Fsrchnum.htm&r=1&f=G&l=50&s1=6045804.PN.&OS=PN/6045804&RS=PN6045804
http://www.journals.uchicago.edu/doi/pdf/10.1086/313920
http://www.amazon.com/Lyme-Disease-Key-Diseases-1/dp/0943126584/ref=sr_1_1?ie=UTF8&s=books&qid=1274807350&sr=1-1F8&s=books&qid=1274807350&sr=1-1
We lost all those years in discovery in the mechanisms of all major chronic diseases and cancer through the years of 1995 to 2008 from when the likes of Yale, Mayo Clinic's Dave Persing (later went to Corixa), and Allen Steere decided to lie about the outcomes of LYMErix, and Gary Wormser and Lenny Sigal decided to lie about the outcome of ImmuLyme.
WHY DID WE LET THIS HAPPEN?
The EVIDENCE says we should treat Chronic Lyme as a chronic spirochetal infection (the treatment of Q fever and tuberculosis with antibiotics could take years and this is well-known) and as some version of some Imitators(s), be it the MS version, the Lupus version, or whatever. We do not know how one treats badly cloned B cells. It could be in the end, some sort of leukophoresis or stem cell transfer of new B cells
"B cells as under-appreciated mediators of non-auto-immune inflammatory
disease."
http://www.ncbi.nlm.nih.gov/pubmed/20382544
"B lymphocytes play roles in many auto-immune diseases characterized by
unresolved inflammation, and B cell ablation is proving to be a relatively safe, effective treatment for such diseases. B cells function, in part, as important sources of regulatory cytokines in auto-immune disease, but B cell cytokines also play roles in other non-auto-immune inflammatory diseases. B cell ablation may therefore benefit inflammatory disease patients in addition to its demonstrated efficacy in auto-immune disease.
Current ablation drugs clear both pro- and anti-inflammatory B cell subsets, which may unexpectedly exacerbate some pathologies. This possibility argues that a more thorough understanding of B cell function in human inflammatory disease is required to safely harness the clinical promise of B cell ablation. Type 2 diabetes (T2D) and periodontal disease (PD) are two inflammatory diseases characterized by little autoimmunity.
These diseases are linked by coincident presentation and alterations in
toll-like receptor (TLR)-dependent B cell cytokine production, which may
identify B cell ablation as a new therapy for co-affected individuals. Further
analysis of the role B cells and B cell cytokines play in T2D, PD and other
inflammatory diseases is required to justify testing B cell depletion therapies
on a broader range of patients." Copyright 2010 Elsevier Ltd. All rights
reserved.
We MUST be an advocate of our own health.
US Doctors are not going to do it for you.
You know what you will tolerate or not.
Inform yourself about Pam3Cys; tell physicians to research it for themselves.
Do it for you and the ones you love.
YOU CAN BEAT LYME!
*
Thank you for being here and learning the truth behind
bacterial infections that are sweeping our globe~
*
Signed ~ Concerned Citizens